Cell Apoptosis of Monocyte-Phagocyte System and Mechanisms for Its Regulation
Keywords:apoptosis, homeostasis, monocyte-phagocyte system, products of embryofetoplacental complex, neuroimmunoendocrine block
Apoptosis was established to play a significant role in maintaining cell and tissue homeostasis. Data on cell sensitivity of monocyte-phagocyte system to apoptosis induction are contradictory, therefore of interest is to reveal the way of realising in them the mechanism of apoptotic program and factors affecting apoptotic process in cells of this system. Nowadays there has been actual the search for new ways using which we could control apoptotic processes, as well as correct the balance of pro- and anti-apoptotic molecule content. The most perspective products with such an ability are those of embryofetoplacental complex since they harmonise the mechanisms of interactions between cells in structures of neuroimmunoendocrine block, responsible for maintaining bodyhomeostasis in a recipientâ€™s organism.
How to Cite
Copyright (c) 2020 A. N. Goltsev, E. E. Yampolskaya
This work is licensed under a Creative Commons Attribution 4.0 International License.
Authors who publish with this journal agree to the following terms:
- Authors retain copyright and grant the journal right of first publication with the work simultaneously licensed under a Creative Commons Attribution License that allows others to share the work with an acknowledgement of the work's authorship and initial publication in this journal.
- Authors are able to enter into separate, additional contractual arrangements for the non-exclusive distribution of the journal's published version of the work (e.g., post it to an institutional repository or publish it in a book), with an acknowledgement of its initial publication in this journal.
- Authors are permitted and encouraged to post their work online (e.g., in institutional repositories or on their website) prior to and during the submission process, as it can lead to productive exchanges, as well as earlier and greater citation of published work (See The Effect of Open Access).